Revealing the pathomechanism of chronic inflammation at single-cell molecular level caused by alpha galactosidase and beta glucocerebrosidase lysosomal enzyme dysfunctions  Page description

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Details of project

  
Identifier
138521
Type PD
Principal investigator Kovács, Árpád Ferenc
Title in Hungarian Az alfa galaktozidáz és béta glükocerebrozidáz lizoszomális enzimdiszfunkciók okozta krónikus gyulladás egy-sejt szintű molekuláris patomechanizmusának vizsgálata
Title in English Revealing the pathomechanism of chronic inflammation at single-cell molecular level caused by alpha galactosidase and beta glucocerebrosidase lysosomal enzyme dysfunctions
Keywords in Hungarian alfa galaktozidáz, béta glükocerebrozidáz, CRISPR-Cas9, FACS, scRNA-Seq
Keywords in English alpha galactosidase, beta glucocerebrosidase, CRISPR-Cas9, FACS, scRNA-Seq
Discipline
Immunology (Council of Medical and Biological Sciences)35 %
Ortelius classification: Immunology
Haematology, immunology, thrombosis, infectious diseases (Council of Medical and Biological Sciences)35 %
Ortelius classification: Haematology
Pediatrics, perinatology (Council of Medical and Biological Sciences)30 %
Panel Cellular and Developmental Biology
Department or equivalent 2nd Dept. of Pediatrics (Semmelweis University)
Starting date 2021-09-01
Closing date 2024-08-31
Funding (in million HUF) 26.999
FTE (full time equivalent) 2.50
state running project




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